Sickle cell disease literature review

Short essay description
The substitution of one amino acid in the hemoglobin molecule results in sickle hemoglobin. As a result, RBCs sickle in low oxygen states causing occlusion of blood vessels, increased viscosity, and inflammation. These RBCs are prematurely removed from the circulation, resulting in a chronic hemolytic anemia. With newborn screening and early treatment, the death rate among children with SCD has declined.
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Sickle cell disease pain management in adolescents: a literature review

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Complications and Treatments of Sickle Cell Disease | CDC

This study evaluated the prevalence rate of vaso-occlusive crisis VOC episodes, rates of uncomplicated and complicated VOC episodes, and the primary reasons for emergency room ER visits and inpatient admissions for sickle cell disease SCD patients. The date of the first observed SCD claim was designated as the index date. Patient demographics, baseline clinical characteristics, the rate of uncomplicated and complicated VOC VOC with concomitant SCD complications episodes, and reasons for ER visits and inpatient stays were analyzed descriptively. A total of 8, patients were included in the analysis, with a median age of 30 years. The average follow-up period was 2. The rate of VOC episodes anytime in the follow-up was 3. During the first-year follow-up period, an average of 2.
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Sickle-cell disease and the heart: review of the current literature

In patients with sickle cell disease, the molecules of mutated hemoglobin HbSS have a propensity to accumulate and coalesce when starved of oxygen. This accumulation in the red blood cell causes the cell to elongate and stiffen, developing into the sickle or crescent shape that is characteristically seen on a blood smear. These changes can lead to occlusion of the microvasculature and cause subsequent disruption of blood circulation, which may lead to transient ischemia of the affected tissues. Although the exact cause of the pain is not fully understood, it is suggested that the ischemia caused by the vaso-occlusion in sickle cell disease triggers a release of inflammatory mediators, which then activate afferent nerve fibers in the surrounding tissue.
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Published: September 1, Published: June 15, You can help by reading and sharing our resources with friends and family. Published: January 21,
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